Makoto KAWATANI
Makotoo KAWATANI
(Ph.D, Senior Research Scientist)
- INTEREST OF MY RESEARCH
- Microorganisms often produce useful compounds for us. I have screened such compounds, especially anticancer compounds, and have studied their mode of action. I want to contribute to development of new drugs through chemical biology research.
- EDUCATION
-
- 1994-1998
- Faculty of Science and Technology, Keio University
- 2001-2003
- JSPS research fellowships for young scientists
- 1998-2003
- Graduate School of Science and Technology, Keio University
- DEGREES
-
- B.Sc. (1998)
- Keio University
- Dr. Science (2003)
- Keio University
- APPOINTMENTS
-
- 2003-2004
- Postdoctoral Researcher, Antibiotics Laboratory, RIKEN
- 2004-2007
- Special Postdoctoral Researcher, Antibiotics Laboratory, RIKEN
- 2007-2009
- Postdoctoral Researcher, Antibiotics Laboratory, RIKEN
- 2009-2012
- Research Scientist, Antibiotics Laboratory, RIKEN
- 2012-2015
- Senior Research Scientist, Chemical Biology Core Facility, RIKEN ASI
- 2015.Apr.-
- Senior Research Scientist, Chemical Biology Research Group, RIKEN CSRS
- AWARDS
-
- The Excellent Paper Award Published in Bioscience, Biotechnology, & Biochemistry (2007)
- Best Presentation Award, 2007 Annual Meeting of the Japanese Association for Molecular Target Therapy of Cancer (2007)
- Research Incentive Award of RIKEN (2009)
- The Excellent Paper Award Published in Bioscience, Biotechnology, & Biochemistry (2009)
- The Research Incentive Award of the Japanese Association for Molecular Target Therapy of Cancer (2009)
- Japanese Cancer Association Incitement Award (2012)
- ACADEMIC ACTIVITIES
-
- Japanese Cancer Association
- The Japanese Association for Molecular Target Therapy of Cancer (Councilor)
- Japan Society for Bioscience, Biotechnology, and Agrochemistry
- Japanese Society for Chemical Biology
- Oncology Research (Editorial Board Member)
- MY HOBBY
- Football, Badminton
- LIST OF SELECTED PUBLICATIONS
-
- Kawamura T.*, Kawatani M.*, Muroi M., Kondoh Y., Futamura Y., Aono H., Tanaka M., Honda K., and Osada H. Proteomic profiling of small-molecule inhibitors reveals dispensability of MTH1 for cancer cell survival. Sci. Rep. 6, 26521 (2016) *, equal contribution
- Kawatani M., Fukushima Y., Kondoh Y., Honda K., Sekine T., Yamaguchi Y., Taniguchi N., and Osada H. Identification of matrix metalloproteinase inhibitors by chemical arrays. Biosci. Biotechnol. Biochem. 79, 1597-1602 (2015)
- Kawatani M., and Osada H. Affinity-based target identification for bioactive small molecules. MedChemComm 5, 277-287 (2014) Review Article
- Futamura Y., Kawatani M., Muroi M., Aono H., Nogawa T., and Osada H. Molecular target identification of a novel fungal metabolite, pyrrolizilactone, by phenotypic profiling systems. Chembiochem 14, 2456-2463 (2013)
- Futamura Y.*, Kawatani M.*, Kazami S., Tanaka K., Muroi M., Shimizu T., Tomita K., Watanabe N., and Osada H. Morphobase, an encyclopedic cell morphology database, and its use for drug target identification. Chem. Biol. 19, 1620-1630 (2012) *, equal contribution
- Kawatani M., Takayama H., Muroi M., Kimura S., Maekawa T., and Osada H. Identification of a small-molecule inhibitor of DNA topoisomerase II by proteomic profiling. Chem. Biol. 18, 743-751 (2011)
- Kawatani M., and Osada H. Osteoclast-targeting small molecules for the treatment of neoplastic bone metastases. Cancer Sci. 100, 1999-2005 (2009) Review Article
- Kawatani M., Okumura H., Honda K., Kanoh N., Muroi M., Dohmae N., Takami M., Kitagawa M., Futamura Y., Imoto M., and Osada H. The identification of an osteoclastogenesis inhibitor through the inhibition of glyoxalase I. Proc. Natl. Acad. Sci. USA 105, 11691-11696 (2008)
- Woo J.-T.*, Kawatani M.*, Kato M., Shinki T., Yonezawa T., Kanoh N., Nakagawa H., Takami M., Lee K. H., Stern P. H., Nagai K., and Osada H. Reveromycin A, an agent for osteoporosis, inhibits bone resorption by inducing apoptosis specifically in osteoclasts. Proc. Natl. Acad. Sci. USA 103, 4729-4734 (2006) *, equal contribution
- Kawatani M., and Imoto M. Deletion of the BH1 domain of Bcl-2 accelerates apoptosis by acting in a dominant negative fashion. J. Biol. Chem. 278, 19732-19742 (2003)